The Relationship Between Onset of Blood Lactate Accumulation, Critical Velocity, and Maximal Lactate Steady State in Soccer Players

2005 ◽  
Vol 19 (2) ◽  
pp. 364 ◽  
Author(s):  
Benedito Sérgio Denadai ◽  
Euripedes Barsanulfo Gonçalves Gomide ◽  
Camila Coelho Greco
Keyword(s):  
Steady State ◽  
Blood Lactate ◽  
Critical Velocity ◽  
Soccer Players ◽  
Maximal Lactate Steady State ◽  
Lactate Accumulation ◽  
Blood Lactate Accumulation ◽  
The Relationship

Threshold for muscle lactate accumulation during progressive exercise

1989 ◽  
Vol 66 (6) ◽  
pp. 2710-2716 ◽  
Author(s):  
J. Chwalbinska-Moneta ◽  
R. A. Robergs ◽  
D. L. Costill ◽  
W. J. Fink
Keyword(s):  
Blood Lactate ◽  
Bicycle Ergometer ◽  
Exercise Tests ◽  
Muscle Lactate ◽  
Lactate Accumulation ◽  
Ergometer Exercise ◽  
Progressive Exercise ◽  
Blood Lactate Accumulation ◽  
Highly Correlated ◽  
The Relationship

The purpose of this study was to investigate the relationship between muscle and blood lactate concentrations during progressive exercise. Seven endurance-trained male college students performed three incremental bicycle ergometer exercise tests. The first two tests (tests I and II) were identical and consisted of 3-min stage durations with 2-min rest intervals and increased by 50-W increments until exhaustion. During these tests, blood was sampled from a hyperemized earlobe for lactate and pH measurement (and from an antecubital vein during test I), and the exercise intensities corresponding to the lactate threshold (LT), individual anaerobic threshold (IAT), and onset of blood lactate accumulation (OBLA) were determined. The test III was performed at predetermined work loads (50 W below OBLA, at OBLA, and 50 W above OBLA), with the same stage and rest interval durations of tests I and II. Muscle biopsies for lactate and pH determination were taken at rest and immediately after the completion of the three exercise intensities. Blood samples were drawn simultaneously with each biopsy. Muscle lactate concentrations increased abruptly at exercise intensities greater than the “below-OBLA” stage [50.5% maximal O2 uptake (VO2 max)] and resembled a threshold. An increase in blood lactate and [H+] also occurred at the below-OBLA stage; however, no significant change in muscle [H+] was observed. Muscle lactate concentrations were highly correlated to blood lactate (r = 0.91), and muscle-to-blood lactate ratios at below-OBLA, at-OBLA, and above-OBLA stages were 0.74, 0.63, 0.96, and 0.95, respectively.(ABSTRACT TRUNCATED AT 250 WORDS)

Effects of hyperoxia on skeletal muscle carbohydrate metabolism during transient and steady-state exercise

2005 ◽  
Vol 98 (1) ◽  
pp. 250-256 ◽  
Author(s):  
Trent Stellingwerff ◽  
Lee Glazier ◽  
Matthew J. Watt ◽  
Paul J. LeBlanc ◽  
George J. F. Heigenhauser ◽  
...  
Keyword(s):  
Skeletal Muscle ◽  
Steady State ◽  
Blood Lactate ◽  
Active Form ◽  
Carbohydrate Oxidation ◽  
Muscle Lactate ◽  
Carbohydrate Utilization ◽  
Lactate Accumulation ◽  
Substrate Phosphorylation ◽  
Blood Lactate Accumulation

This study compared the effects of inspiring either a hyperoxic (60% O2) or normoxic gas (21% O2) while cycling at 70% peak O2 uptake on 1) the ATP derived from substrate phosphorylation during the initial minute of exercise, as estimated from phosphocreatine degradation and lactate accumulation, and 2) the reliance on carbohydrate utilization and oxidation during steady-state cycling, as estimated from net muscle glycogen use and the activity of pyruvate dehydrogenase (PDH) in the active form (PDHa), respectively. We hypothesized that 60% O2 would decrease substrate phosphorylation at the onset of exercise and that it would not affect steady-state exercise PDH activity, and therefore muscle carbohydrate oxidation would be unaltered. Ten active male subjects cycled for 15 min on two occasions while inspiring 21% or 60% O2, balance N2. Blood was obtained throughout and skeletal muscle biopsies were sampled at rest and 1 and 15 min of exercise in each trial. The ATP derived from substrate-level phosphorylation during the initial minute of exercise was unaffected by hyperoxia (21%: 52.2 ± 11.1; 60%: 54.0 ± 9.5 mmol ATP/kg dry wt). Net glycogen breakdown during 15 min of cycling was reduced during the 60% O2 trial vs. 21% O2 (192.7 ± 25.3 vs. 138.6 ± 16.8 mmol glycosyl units/kg dry wt). Hyperoxia had no effect on PDHa, because it was similar to the 21% O2 trial at rest and during exercise (21%: 2.20 ± 0.26; 60%: 2.25 ± 0.30 mmol·kg wet wt−1·min−1). Blood lactate was lower (6.4 ± 1.0 vs. 8.9 ± 1.0 mM) at 15 min of exercise and net muscle lactate accumulation was reduced from 1 to 15 min of exercise in the 60% O2 trial compared with 21% (8.6 ± 5.1 vs. 27.3 ± 5.8 mmol/kg dry wt). We concluded that O2 availability did not limit oxidative phosphorylation in the initial minute of the normoxic trial, because substrate phosphorylation was unaffected by hyperoxia. Muscle glycogenolysis was reduced by hyperoxia during steady-state exercise, but carbohydrate oxidation (PDHa) was unaffected. This closer match between pyruvate production and oxidation during hyperoxia resulted in decreased muscle and blood lactate accumulation. The mechanism responsible for the decreased muscle glycogenolysis during hyperoxia in the present study is not clear.

Relationship of Maximal Lactate Steady State to Performance at Fixed Blood Lactate Reference Values in Children

1991 ◽  
Vol 3 (4) ◽  
pp. 333-341 ◽  
Author(s):  
Joanne R. Williams ◽  
Neil Armstrong
Keyword(s):  
Steady State ◽  
Blood Lactate ◽  
Reference Values ◽  
Treadmill Exercise ◽  
Reference Value ◽  
Aerobic Performance ◽  
Reference Level ◽  
Maximal Lactate Steady State ◽  
Relationship Of ◽  
The Relationship

This investigation set out to estimate exercise intensity and blood lactate corresponding to the maximal lactate steady state (MLaSS) and also examined the relationship between performance at the MLaSS with performance at fixed blood lactate reference values of 2.5 and 4.0 mmol•1−1. Cardiopulmonary responses at peak treadmill exercise and blood lactate reference values were measured in 10 boys and 8 girls ages 13-14 years. The 2.5 mmol•11 reference value represented 84±7% peak VO2 in boys and 82±6% peak VO2 in girls. Corresponding values at the 4.0 mmol•1−1 level were 93±6% and 90±5% peak VO2. MLaSS occurred at 77±7% peak VO2 in boys and 76±7% peak VO2 in girls. Blood lactate at the MLaSS was 2.1±0.5 mmol•l−1 in boys and 2.3±0.6 mmol•l−1 in girls. Cardiopulmonary and heart rate responses at the MLaSS were not significantly different from corresponding responses at the 2.5 mmol•l−1 reference value. In contrast, cardiopulmonary responses at the 4.0 mmol•l−1 reference level were significantly higher than those at the MLaSS. These data indicate that a 2.5 mmol•l−1 criterion for assessing aerobic performance in children may be the most appropriate.

scholarly journals Exercise intensity during official soccer matches

2016 ◽  
Vol 18 (6) ◽  
pp. 621 ◽  
Author(s):  
Daniel Barbosa Coelho ◽  
Rodney Coelho da Paixão ◽  
Emerson Cruz de Oliveira ◽  
Lenice Kappes Becker ◽  
João Batista Ferreira-Júnior ◽  
...  
Keyword(s):  
Heart Rate ◽  
Blood Lactate ◽  
Field Test ◽  
Exercise Intensity ◽  
Intermediate Phase ◽  
Age Groups ◽  
Soccer Players ◽  
Blood Lactate Accumulation ◽  
Intensity Of Exercise ◽  
In The Beginning

DOI: http://dx.doi.org/10.5007/1980-0037.2016v18n6p621 The aims of the study were: 1) to analyze the exercise intensity in several phases (six phases of 15 min) of soccer matches; 2) to compare the match time spent above anaerobic threshold (AT) between different age groups (U-17 and U-20); and 3) to compare the match time spent above AT between players’ positions (backs, midfielders, forwards and wingabcks). Forty-four male soccer players were analyzed. To express players’ effort, the heart rate (HR) was continuously monitored in 29 official matches. Further, HR corresponding to the intensity at the onset of blood lactate accumulation (OBLA) was obtained in a field test. The highest exercise intensity during match was observed in the 15-30 min period of the first half (p< 0.05). Match time spent above AT was not different between players from U-17 and U-20. In the comparison among players’ positions, wingbacks showed lower time above AT (p< 0.05) than players of other positions. The intensity of effort is higher in the 15 to 30 minutes of play (intermediate phase), probably because the players are more rested in the beginning and wearing out is progressive throughout the game. It is also noteworthy that the intensity of exercise (HR and time above AT) of wingbacks was lower, probably because they usually are required to run a larger number of sprints and need more time below the AT to recover. 

Sign in / Sign up

Export Citation Format

Share Document